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Research to date suggests that this attack is primarily aimed at myelin and oligodendrocytes, cells responsible for making and maintaining myelin.
Recurring episodes of MS can cause many scars to form in the central nervous system (CNS) where there has been breakdown of myelin, the insulating material that covers the nerve fibres. This damage can result in impairment of motor, sensory and/or cognitive functions to a greater or lesser extent.
The CNS includes the brain, optic nerves and spinal cord, act as the body’s messenger system.
Myelin is the fatty covering (white matter) that serves as insulation, like the plastic coating on an electrical wire. This helps the efficient transmission of nerve impulses (messages) between the brain and other parts of the body.
In MS, a process called demyelination causes scar tissue or sclerotic plaques (lesions) to form along the myelin sheath. These interfere with the conduction of nerve impulses within the CNS, which causes the myriad of symptoms people may experience over the course of MS.
In addition research indicates that damage may also occur to the nerve fibres (axons) themselves. Because this damage is seen in active, inflammatory lesions, researchers have suggested that the immune attack in MS may also target nerve cells directly, not just the myelin and oligodendrocyte cells. Evidence indicates that this damage may occur early in the course of MS, reinforcing the importance of early treatment.
While there is no way to predict with any accuracy how a person’s MS will progress, four basic clinical patterns have been defined:
References: MS Australia
Knowledge is Power Volume 1.
Read more at: MS Trust